VTI1B (NM_006370) Human 3' UTR Clone

CAT#: SC203291

3`UTR clone of vesicle transport through interaction with t-SNAREs homolog 1B (yeast) (VTI1B) for miRNA target validation


Reconstitution Protocol

USD 560.00

4 Weeks*

Size
    • 10 ug

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Specifications

Product Data
Vector pMirTarget
Species Human
Transfection Reporter RFP
Assay Reporter Luciferase
E. coli Selection Kanamycin (25 ug/mL)
Mammalian Cell Selection Neomycin
Symbol VTI1B
Synonyms v-SNARE; VTI1; VTI1-LIKE; vti1-rp1; VTI1L; VTI2
ACCN NM_006370
Insert Size 228
Sequence Data
>SC203291 3'UTR clone of NM_006370
The sequence shown below is from the reference sequence of NM_006370. The complete sequence of this clone may contain minor differences, such as SNPs. Red=Cloning site Blue=Stop Codon


CAATTGGCAGAGCTCAGAATTCAAGCGATCGC

ATTCTTTCGCAGCCATTGAACTTCTATAGGGAAGGGTTTGTGGACCAGAACTTTGACCTTGTGAATGCAT
GATGTTAGGGATGTGGATAGAATAAGCATATTGCTGCTGTGGGCTGACAGTTCAAGGATGCACTGTATAG
CCAGGCTGTGGGAGGAGGGAGGAAAGATGAAAAACCACTTAAATGTGAAGGAACAACAGCAACAAGACCA
GTATGATATACCAAGGTA

ACGCGTAAGCGGCCGCGGCATCTAGATTCGAAGAAAATGACCG
Restriction Sites SgfI-MluI     
OTI Disclaimer Our molecular clone sequence data has been matched to the sequence identifier above as a point of reference. Note that the complete sequence of this clone is largely the same as the reference sequence but may contain minor differences , e.g., single nucleotide polymorphisms (SNPs).
Reference Data
RefSeq NM_006370.2
Summary V-SNARE that mediates vesicle transport pathways through interactions with t-SNAREs on the target membrane. These interactions are proposed to mediate aspects of the specificity of vesicle trafficking and to promote fusion of the lipid bilayers. May be concerned with increased secretion of cytokines associated with cellular senescence. [UniProtKB/Swiss-Prot Function]
Locus ID 10490

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*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.