RABL2A Human Gene Knockout Kit (CRISPR)
CAT#: KN402356
RABL2A - KN2.0, Human gene knockout kit via CRISPR, non-homology mediated.
KN2.0 knockout kit validation
KN402356 is the updated version of KN202356.
USD 1,290.00
2 Weeks*
Size
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Specifications
Product Data | |
Format | 2 gRNA vectors, 1 linear donor |
Donor DNA | EF1a-GFP-P2A-Puro |
Symbol | RABL2A |
Locus ID | 11159 |
Disclaimer | The kit is designed based on the best knowledge of CRISPR technology. The system has been functionally validated for knocking-in the cassette downstream the native promoter. The efficiency of the knock-out varies due to the nature of the biology and the complexity of the experimental process. |
Reference Data | |
RefSeq | NM_001306158, NM_001306159, NM_001306160, NM_001306161, NM_007082, NM_013412, NM_001354405, NM_001354406, NM_001354407, NM_001354408, NM_001354409, NM_001354410, NM_001354412, NM_001354413, NM_001354414, NM_001354416, NM_001354417, NM_001354418, NM_001354419, NM_001354421, NM_001354423, NM_001354424, NM_001354425, NM_001354426, NM_001354427, NM_001354428, NR_148879, NR_148880, NR_148881, NR_148882 |
Synonyms | FLJ78724; MGC117180 |
Summary | This gene is a member of the RAB gene family which belongs to the RAS GTPase superfamily. The proteins in the family of RAS-related signaling molecules are small GTP-binding proteins that play important roles in the regulation of exocytotic and endocytotic pathways. This gene maps to the site of an ancestral telomere fusion event and may be a subtelomeric gene. Alternative splicing results in multiple transcript variants encoding different isoforms. [provided by RefSeq, Apr 2015] |
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Other Versions
SKU | Description | Size | Price |
---|---|---|---|
GA107681 | RABL2A CRISPRa kit - CRISPR gene activation of human RAB, member of RAS oncogene family like 2A |
USD 1,290.00 |
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*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen
complexities in the preparation of your product. International customers may expect an additional 1-2 weeks
in shipping.